Friday October 24
Boston Cure Project
Researchers from the University of Maryland trying to understand the
therapeutic mechanisms of Copaxone (glatiramer acetate) found that it may
stimulate production of a neuroprotective molecule. Using blood samples
drawn from MS patients on Copaxone, they studied production of brain-derived
neurotrophic factor (BDNF), a protein that enhances neural development,
protects mature neurons, and promotes remyelination. They found that Copaxone-reactive
T cells produced more BDNF than did T cells reactive to other antigens.
So this suggests that one of the ways in which Copaxone may help people
with MS is through the stimulation of agents that protect damaged axons
and facilitate remyelination.
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